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TSRI 19th Annual Poster Session



  • Members of our interdisciplinary neuro-oncology team recently represented Children's Hospital Los Angeles at the International Symposium on Pediatric Neuro-Oncology (ISPNO) in Singapore. The team presented nine abstracts covering a variety of topics, from iron overload in brain tumor patients to new biomarkers for recognizing a specific gene mutation in pediatrics giloblastomas. Held every two years, the ISPNO conference attracts delegates from all over the world to discuss innovative research, advances in pediatric neuro-oncology and new surgical treatments.

  • Kathleen Ruccione, PhD, MPH, RN and Kathleen Meeske, PhD, received the Cancer Nursing 2014 Writing Award, recognizing their paper as one of the two best-written in Cancer Nursing. The paper, “Adolescents’ Psychosocial Health-Related Quality of Life Within 6 Months After Cancer Treatment Completion,” examined the psychosocial health-related quality of life (HRQOL) in adolescents who recently completed cancer treatment. Since battling a life-threatening illness can trigger negative psychosocial symptoms such as pain, fatigue, depression and posttraumatic stress, Ruccione, Meeske and colleagues recommend that HRQOL be assessed during the early post-cancer treatment phase to maximize healthy survival.

  • Jennifer C. Miller, MD, PhD, has been named the 2014 Fellow of the CHLA George Donnell Society for Pediatric Scientists. As a Donnell Fellow, Miller will receive mentorship, educational opportunities and administrative support for grant and manuscript submissions. This Society, led by Steven Mittelman, MD, PhD, is dedicated to improving the health of children and provides the infrastructure and guidance to train pediatric scientists to perform innovative and impactful research.


  • How a Single, Genetic Change Causes Retinal Tumors in Young Children
  • David E. Cobrinik, MD, PhD, of The Vision Center at Children’s Hospital Los Angeles (CHLA), together with colleagues at Memorial Sloan-Kettering Cancer Center, has answered the long-standing question of why mutations to the RB1 gene primarily cause tumors of the retina and not of other cell types. 



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